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Difference between revisions of "Talk:MiP2014"

From Bioblast
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|ย  [[Ali SS|Ali Sameh S]]||Zewail City for Science and Technology, ET||Oxygen-consuming and ROS-producing activities in synaptosomes.
|ย  [[Ali SS|Ali Sameh S]]||Zewail City for Science and Technology, ET||Oxygen-consuming and ROS-producing activities in synaptosomes.
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|ย  [[Borutaite_Vilmante|Borutaite Vilmante]]||Lithuanian University of Health Sciences, Kaunas, LT||Controversies in testing integrity of mitochondrial membranes.
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|ย  Brown David||East Carolina University, NC, US||
|ย  Brown David||East Carolina University, NC, US||

Revision as of 15:18, 5 March 2014

10th MiPconference - MiP2014: Joint IUBMB/MiP Symposium on Mitochondrial Physiology - a Point/Counterpoint Meeting

The Mitochondrial Physiology Society, Obergurgl, Tyrol, Austria. 2014-Sep-08 to 12

>>> Book of Abstracts Open Access

MiP2014 Abstracts filtered by MipMaps

All abstracts in the MiPMap

Keynotes


MiP2014 participants

Name Institution Preliminary title
Ali Sameh S Zewail City for Science and Technology, ET Oxygen-consuming and ROS-producing activities in synaptosomes.
Borutaite Vilmante Lithuanian University of Health Sciences, Kaunas, LT Controversies in testing integrity of mitochondrial membranes.
Brown David East Carolina University, NC, US
Dohlmann Tine University of Copenhagen, DK
Hepple Russel T. McGill University, Montreal, CA Mitochondrial involvement in aging muscle: cause and effect(or).
Kraunsoe Regitze University of Copenhagen, DK
Koopman Werner J Radboud University Nijmegen Medical Centre, NL Redox regulation of mitochondrial dynamics.
Larsen Steen University of Copenhagen, DK Normalization of respiration.
Picard Martin University of Pennsylvania, PA, US
Pecina Peter Academy of Sciences of the Czech Republic, Prague, CZ The use of lymphocytes for diagnostics of mitochondrial disorders.
Purhonen Janne University Helsinki, FI
Ristow Michael ETH Zurich, CH Promoting health and lifespan by increasing mitochondrial ROS formation.
Stiban Johnny Birzeit University, PS Very long chain ceramides interfere with C16-ceramide-induced channel formation: A plausible mechanism for regulating the initiation of intrinsic apoptosis.

Further MiPMap pages

MiPsociety
Contact: Gerhard Krumschnabel


Post-conference Workshop