Scatena Roberto

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COST Action CA15203 (2016-2021): MitoEAGLE
Evolution-Age-Gender-Lifestyle-Environment: mitochondrial fitness mapping

Scatena Roberto

MitoPedia topics: EAGLE

COST: Member COST WG1: WG1 COST WG2: WG2 COST WG3: WG3 COST WG4: WG4 COST Mentor: Mentor

MC Member - Management Committee MitoEAGLE

Name	Scatena Roberto, Prof. PhD
Institution	Department of Diagnostics and Laboratory Medicine, Institute of Biochemistry and Clinical Biochemistry, Polyclinic A. Gemelli - Catholic University, IT
Address	Largo A. Gemelli 8, 00168
City	Rome
State/Province
Country	Italy
Email	[email protected]
Weblink
O2k-Network Lab	IT Rome Scatena R

New Open Access Journal

Dr. R. Scatena is the editor of 'Cancer Stem Cells'.

Labels: Field of research: Basic

Publications

	Published	Reference
BEC 2020.1 doi10.26124bec2020-0001.v1	2020	Gnaiger E et al ― MitoEAGLE Task Group (2020) Mitochondrial physiology. https://doi.org/10.26124/bec:2020-0001.v1
Bottoni 2019 Adv Exp Med Biol	2019	Bottoni P, Scatena R (2019) Mitochondrial metabolism in cancer. A tangled topic. Which role for proteomics? Adv Exp Med Biol 1158:1-16.
Scatena 2019 Curr Med Chem	2019	Scatena R, Bottoni P (2019) The tangled mitochondrial metabolism in cancer: An innovative pharmacological approach. Curr Med Chem 27:2106-17.
Tesori 2015 Sci Rep	2015	Tesori V, Piscaglia AC, Samengo D, Barba M, Bernardini C, Scatena R, Pontoglio A, Castellini L, Spelbrink JN, Maulucci G, Puglisi MA, Pani G, Gasbarrini A (2015) The multikinase inhibitor Sorafenib enhances glycolysis and synergizes with glycolysis blockade for cancer cell killing. Sci Rep 5:9149.
Scatena 2012 Adv Exp Med Biol	2012	Scatena R, Bottoni P, Giardina B (eds) (2012) Advances in mitochondrial medicine. Adv Exp Med Biol 942:p461.

Abstracts

	Published	Reference
Scatena 2018 MiP2018	2018	No abstract.
Scatena 2017 Abstract MITOEAGLE Barcelona	2017	Mitochondria and cancer stem cells in hepatocarcinoma. An illustrative example of a tangled methodological approach.
Scatena 2017 MITOEAGLE Obergurgl	2017	Cancer stem cell differentiation induced by PPAR ligands. Which role for an inhibition of mitochondrial respiratory chain?

MitoEAGLE Feedback

Specifically, we are studying mitochondrial metabolism in cancer cells. At this aim we are also participating to mt-HPP project, an Italian consortium whose target is a definition of mitochondrial proteome in health and disease. Briefly, my specific activity is to compare mitochondrial functional status with the mitochondrial proteome picture in different human cell line. I do think that a "contact" between these two important initiatives should be useful for both.

Moreover, to stress the potential translational applications of research on bioenergetics in general and on mitochondria in particular (as also recently highlighted by Paolo Bernardi during EBEC 2016), I would like to suggest to begin to think to a careful pathophysiological classification of all mitochondriopathies (inborn and acquired, primary and secondary, ECR of metabolic related and so on) to reinforce the concept of a “mitochondrial medicine”, concept, until now, too often neglected with dramatic consequences. - Scatena Roberto (2016).

Participated at