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Difference between revisions of "Escande CC"

From Bioblast
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{{Person
{{Person
|lastname=Escande Castro ย 
|lastname=Escande Castro
|firstname=Carlos
|firstname=Carlos
|title=PhD
|title=PhD
|mailaddress=
|address=Anesthesia Dept.
|address=Anesthesia Dept.


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55905 MN, USA
55905 MN, USA
}}
}}
{{Labelingperson}}
In our lab we are interested in how protein acetylation dynamics in the mitochondria affects ist function, and how these acetylation pathways are regulated.
In our lab we are interested in how protein acetylation dynamics in the mitochondria affects ist function, and how these acetylation pathways are regulated.

Revision as of 16:20, 8 March 2016

Name Escande Castro Carlos, PhD
Institution
Address Anesthesia Dept.

Mayo Clinic

200 1st ST SW, Rochester

55905 MN, USA,

City
State/Province
Country
Email
Weblink
O2k-Network Lab


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Publications

 PublishedReference
Camacho-Pereira 2016 Cell Metab2016Camacho-Pereira J, Tarragรณ MG, Chini CC, Nin V, Escande C, Warner GM, Puranik AS, Schoon RA, Reid JM, Galina A, Chini EN (2016) CD38 dictates age-related NAD decline and mitochondrial dysfunction through an SIRT3-dependent mechanism. Cell Metab 23:1127-39.
Chini 2014 Clin Cancer Res2014Chini CC, Gonzalez Guerrico A, Nin V, Camacho-Pereira J, Escande C, Barbosa M, Chini EN (2014) Targeting of NAD metabolism in pancreatic cancer cells: potential novel therapy for pancreatic tumors. Clin Cancer Res 20:120-30.
Nin 2012 J Biol Chem2012Nin V, Escande C, Chini CC, Giri S, Camacho-Pereira J, Matalonga J, Lou Z, Chini EN (2012) Role of deleted in breast cancer 1 (DBC1) protein in SIRT1 deacetylase activation induced by protein kinase A and AMP-activated protein kinase. J Biol Chem 287:23489-501.

Abstracts

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In our lab we are interested in how protein acetylation dynamics in the mitochondria affects ist function, and how these acetylation pathways are regulated.