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Kramer Philip A

From Bioblast
Revision as of 12:03, 2 March 2020 by Gnaiger Erich (talk | contribs) (Gnaiger Erich moved page Kramer PA to Kramer Philip A over redirect)
Name Kramer Philip A, Dr.
Institution
Kramer P
Wake Forest Baptist Medical Center, US
Address Medical Center Blvd, 27157
City Winston-Salem
State/Province North Carolina (NC)
Country USA
Email [email protected]
Weblink
O2k-Network Lab US NC Winston-Salem Kramer PA, US NC Winston-Salem Molina AJA, US WA Seattle Marcinek DJ


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Publications

 PublishedReference
Kramer 2014 J Vis Exp2014Kramer PA, Chacko BK, Ravi S, Johnson MS, Mitchell T, Darley-Usmar VM (2014) Bioenergetics and the oxidative burst: protocols for the isolation and evaluation of human leukocytes and platelets. J Vis Exp 2014 Mar 27(85). doi: 10.3791/51301.
Kramer 2014 Redox Biol2014Kramer PA, Ravi S, Chacko B, Johnson MS, Darley-Usmar VM (2014) A review of the mitochondrial and glycolytic metabolism in human platelets and leukocytes: implications for their use as bioenergetic biomarkers. Redox Biol 2:206–10.
Chacko 2013 Lab Invest2013Chacko BK, Kramer PA, Ravi S, Johnson MS, Hardy RW, Ballinger SW, Darley-Usmar VM (2013) Methods for defining distinct bioenergetic profiles in platelets, lymphocytes, monocytes, and neutrophils, and the oxidative burst from human blood. Lab Invest 93:690-700.

Abstracts

 PublishedReference
Kramer 2019 Abstract IOC1412019Kramer P, Distefano G, Monte J, Mills A, Edmunds L, Jurczak M, Cummings SR, Kritchevsky S, Newman AB, Hepple RT, Goodpaster B, Coen PM, Molina A (2019) The Study of Muscle, Mobility, and Aging (SOMMA): A multi-institutional respirometry study. Mitochondr Physiol Network 24.02.
Scandalis 2019 Abstract IOC1412019Scandalis LM, Kramer P, Nelson B, Dozier S, Stone J, Kitzman D, Molina AJA (2019) Oxygen consumption of skeletal muscle in heart failure with preserved ejection fraction. Mitochondr Physiol Network 24.02.
Marcinek 2016 Abstract MitoFit Science Camp 20162016Targeting mitochondrial redox stress reveals two phases for reversal of mitochondrial dysfunction in aged mouse skeletal muscle.

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