Difference between revisions of "Kuznetsov 1999 Transplant Proc"
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{{Publication | {{Publication | ||
|title=Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger E (1999) Estimation of mitochondrial damage in heart preservation. Transplant Proc 31: 992. | |title=Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger E (1999) Estimation of mitochondrial damage in heart preservation. Transplant Proc 31:992. | ||
|info=[http://www.ncbi.nlm.nih.gov/pubmed/10083441 PMID: 10083441] | |info=[http://www.ncbi.nlm.nih.gov/pubmed/10083441 PMID: 10083441] | ||
|authors=Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger | |authors=Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger Erich | ||
|year=1999 | |year=1999 | ||
|journal=Transplant Proc | |journal=Transplant Proc | ||
|abstract=STUDIES of mitochondrial bioenergetics yield a quantitative assessment of cell injury after organ storage.<sup>1-4</sup>Β To reduce artifacts of mitochondrial isolation, we optimized high-resolution respirometry with permeabilized cells and muscle fibers for investigation of mitochondrial cold ischemia/reperfusion (CIR) injury. | |abstract=STUDIES of mitochondrial bioenergetics yield a quantitative assessment of cell injury after organ storage.<sup>1-4</sup>Β To reduce artifacts of mitochondrial isolation, we optimized high-resolution respirometry with permeabilized cells and muscle fibers for investigation of mitochondrial cold ischemia/reperfusion (CIR) injury. | ||
|mipnetlab= | |mipnetlab=AT Innsbruck Gnaiger E | ||
|discipline=Mitochondrial Physiology, Biomedicine | |discipline=Mitochondrial Physiology, Biomedicine | ||
}} | }} | ||
{{Labeling | {{Labeling | ||
|organism=Rat | |organism=Rat | ||
|tissues= | |tissues=Heart | ||
|preparations=Permeabilized cells, Permeabilized tissue | |preparations=Permeabilized cells, Permeabilized tissue | ||
|injuries=Ischemia-reperfusion | |||
|topics=Substrate | |||
|couplingstates=OXPHOS | |couplingstates=OXPHOS | ||
| | |instruments=Oxygraph-2k | ||
|discipline=Mitochondrial Physiology, Biomedicine | |discipline=Mitochondrial Physiology, Biomedicine | ||
}} | }} |
Latest revision as of 04:24, 23 November 2021
Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger E (1999) Estimation of mitochondrial damage in heart preservation. Transplant Proc 31:992. |
Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger Erich (1999) Transplant Proc
Abstract: STUDIES of mitochondrial bioenergetics yield a quantitative assessment of cell injury after organ storage.1-4 To reduce artifacts of mitochondrial isolation, we optimized high-resolution respirometry with permeabilized cells and muscle fibers for investigation of mitochondrial cold ischemia/reperfusion (CIR) injury.
β’ O2k-Network Lab: AT Innsbruck Gnaiger E
Labels:
Stress:Ischemia-reperfusion Organism: Rat Tissue;cell: Heart Preparation: Permeabilized cells, Permeabilized tissue
Regulation: Substrate Coupling state: OXPHOS
HRR: Oxygraph-2k