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Difference between revisions of "Kuznetsov 1999 Transplant Proc"

From Bioblast
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|preparations=Permeabilized cells, Permeabilized tissue
|preparations=Permeabilized cells, Permeabilized tissue
|injuries=Ischemia-reperfusion;preservation
|injuries=Ischemia-reperfusion;preservation
|topics=Substrate;glucose;TCA cycle
|topics=Substrate
|couplingstates=OXPHOS
|couplingstates=OXPHOS
|instruments=Oxygraph-2k
|instruments=Oxygraph-2k
|discipline=Mitochondrial Physiology, Biomedicine
|discipline=Mitochondrial Physiology, Biomedicine
}}
}}

Revision as of 15:31, 15 June 2015

Publications in the MiPMap
Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger E (1999) Estimation of mitochondrial damage in heart preservation. Transplant Proc 31:992.

Β» PMID: 10083441

Kuznetsov AV, Brandacher G, Steurer W, Margreiter R, Gnaiger E (1999) Transplant Proc

Abstract: STUDIES of mitochondrial bioenergetics yield a quantitative assessment of cell injury after organ storage.1-4 To reduce artifacts of mitochondrial isolation, we optimized high-resolution respirometry with permeabilized cells and muscle fibers for investigation of mitochondrial cold ischemia/reperfusion (CIR) injury.


β€’ O2k-Network Lab: AT Innsbruck Gnaiger E


Labels:

Stress:Ischemia-reperfusion;preservation"Ischemia-reperfusion;preservation" is not in the list (Cell death, Cryopreservation, Ischemia-reperfusion, Permeability transition, Oxidative stress;RONS, Temperature, Hypoxia, Mitochondrial disease) of allowed values for the "Stress" property.  Organism: Rat  Tissue;cell: Heart  Preparation: Permeabilized cells, Permeabilized tissue 

Regulation: Substrate  Coupling state: OXPHOS 

HRR: Oxygraph-2k