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Lopez MG

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COST Action CA15203 (2016-2021): MitoEAGLE
Evolution-Age-Gender-Lifestyle-Environment: mitochondrial fitness mapping


Lopez MG


MitoPedia topics: EAGLE 

COST: Participant


Name Lopez Manuela, MD, PhD
Institution Departamento de FarmacologĂ­a

Facultad de Medicina

Address C/Arzobispo Morcillo 4, 28029
City Madrid
State/Province
Country Spain
Email [email protected]
Weblink
O2k-Network Lab


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Publications

 PublishedReference
Hernansanz-Agustin 2020 Nature2020Hernansanz-Agustín P, Choya-Foces C, Carregal-Romero S, Ramos E, Oliva T, Villa-Piña T, Moreno L, Izquierdo-Álvarez A, Cabrera-García JD, Cortés A, Lechuga-Vieco AV, Jadiya P, Navarro E, Parada E, Palomino-Antolín A, Tello D, Acín-Pérez R, Rodríguez-Aguilera JC, Navas P, Cogolludo Á, López-Montero I, Martínez-Del-Pozo Á, Egea J, López MG, Elrod JW, Ruíz-Cabello J, Bogdanova A, Enríquez JA, Martínez-Ruiz A (2020) Na+ controls hypoxic signalling by the mitochondrial respiratory chain. Nature 586:287-91.
Hernansanz-Agustin 2019 bioRxiv2019Hernansanz-Agustín P, Choya-Foces C, Carregal-Romero S, Ramos E, Oliva T, Villa-Piña T, Moreno L, Izquierdo-Álvarez A, JCabrera-García JD, Cortés A, Lechuga-Vieco AV, Jadiya P, Navarro E, Parada E, Palomino-Antolín A, Tello D, Acín-Pérez R, Rodríguez-Aguilera JC, Navas P, Cogolludo A, López-Montero I, Martínez-del-Pozo A, Egea J, López MG, Elrod JW, Ruiz-Cabello J, Bogdanova A, Enríquez JA, Martínez-Ruiz A (2019) Mitochondrial Na+ import controls oxidative phosphorylation and hypoxic redox signalling. bioRxiv doi: https://doi.org/10.1101/385690.
Guerra-Alvarez 2015 J Neurochem2015Guerra-Álvarez M, Moreno-Ortega AJ, Navarro E, Fernåndez-Morales JC, Egea J, López MG, Cano-Abad MF (2015) Positive allosteric modulation of alpha-7 nicotinic receptors promotes cell death by inducing Ca2+ release from the endoplasmic reticulum. J Neurochem 133:309-19.
Moreno-Ortega 2015 Aging Cell2015Moreno-Ortega AJ, Buendia I, Mouhid L, Egea J, Lucea S, Ruiz-Nuño A, LĂłpez MG, Cano-Abad MF (2015) CALHM1 and its polymorphism P86L differentially control Ca2+ homeostasis, mitogen-activated protein kinase signaling, and cell vulnerability upon exposure to amyloid ÎČ. Aging Cell 14:1094-102.

Abstracts

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