Wanders RJA

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COST Action CA15203 (2016-2021): MitoEAGLE
Evolution-Age-Gender-Lifestyle-Environment: mitochondrial fitness mapping

 

Wanders RJA

MitoPedia topics: EAGLE 

COST: Member

Name	Wanders Ronald, Prof.
Institution	Lab Genetic Metabolic Diseases, Room F0-223 Academic Medical Center University of Amsterdam
Address	Meibergdreef 9, 1105 AZ
City	Amsterdam
State/Province
Country	The Netherlands
Email	[email protected]
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Publications

	Published	Reference
Van Zutphen 2016 J Hepatol	2016	van Zutphen T, Ciapaite J, Bloks VW, Ackereley C, Gerding A, Jurdzinski A, Allgayer de Moraes R, Zhang L, Wolters JC, Bischoff R, Wanders RJA, Houten SM, Bronte-Tinkew D, Shatseva T, Lewis GF, Groen AK, Reijngoud DJ, Bakker BM, Jonker JW, Kim PK, Bandsma RH (2016) Malnutrition-associated liver steatosis and ATP depletion is caused by peroxisomal and mitochondrial dysfunction. J Hepatol 65:1198-1208.
Wessels 2015 Am J Physiol	2015	Wessels B, van den Broek NM, Ciapaite J, Houten SM, Wanders RJA, Nicolay K, Prompers JJ (2015) Carnitine supplementation in high-fat diet-fed rats does not ameliorate lipid-induced skeletal muscle mitochondrial dysfunction in vivo. Am J Physiol 309(7):E670-8.
Houten 2010 J Inherit Metab Dis	2010	Houten SM, Wanders RJ (2010) A general introduction to the biochemistry of mitochondrial fatty acid β-oxidation. J Inherit Metab Dis 33:469-77.
Van den Broek 2010 FASEB J	2010	van den Broek NM, Ciapaite J, De Feyter HM, Houten SM, Wanders RJA, Jeneson JA, Nicolay K, Prompers JJ (2010) Increased mitochondrial content rescues in vivo muscle oxidative capacity in long-term high-fat-diet-fed rats. FASEB J 24:1354-64.
Oezen 2005 Hum Mol Genet	2005	Oezen I, Rossmanith W, Forss-Petter S, Kemp S, Voigtlander T, Moser-Thier K, Wanders RJA, Bittner RE, Berger J (2005) Accumulation of very long-chain fatty acids does not affect mitochondrial function in adrenoleukodystrophy protein deficiency. Hum Mol Genet 14:1127-37.

Abstracts

	Published	Reference
Wuest 2018 MiP2018a	2018	Reduction of acylcarnitines restores electrophysiological abnormalities in VLCAD deficient hiPSC-cardiomyocytes.