Jaburek M: Difference between revisions
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|firstname=Martin | |firstname=Martin | ||
|title=Dr. | |title=Dr. | ||
|institution=Institute of Physiology, | |institution=[[File:Jaburek_M.JPG|right|150px]] | ||
Institute of Physiology, Academy of Sciences of the Czech Republic, Videnska 1083 | |||
Academy of Sciences of the Czech Republic | |||
|city=Prague | |city=Prague | ||
|country=Czech Republic | |country=Czech Republic | ||
}} | }} | ||
{{Labelingperson | {{Labelingperson | ||
|field of research=Basic | |field of research=Basic | ||
}} | }} | ||
== | == Participated at == | ||
* [ | ::::* [[MiP2014| MiP2014 Obergurgl AT]] | ||
::::* [[MiPNet18.04 IOC76|IOC76 Prague CZ]] |
Revision as of 13:27, 8 February 2018
Name | Jaburek Martin, Dr. |
---|---|
Institution |
Institute of Physiology, Academy of Sciences of the Czech Republic, Videnska 1083 |
Address | , |
City | Prague |
State/Province | |
Country | Czech Republic |
[email protected] | |
Weblink | |
O2k-Network Lab | CZ Prague Jezek P |
Labels:
Field of research: Basic
Publications
Published | Reference | |
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Jezek 2023 Antioxid Redox Signal | 2023 | Ježek P, Jabůrek M, Holendová B, Engstová H, Dlasková A (2023) Mitochondrial cristae morphology reflecting metabolism, superoxide formation, redox homeostasis, and pathology. Antioxid Redox Signal 39:635–83. https://doi.org/10.1089/ars.2022.0173 |
Plecita-Hlavata 2020 Diabetes | 2020 | Plecitá-Hlavatá L, Jabůrek M, Holendová B, Tauber J, Pavluch V, Berková Z, Cahová M, Schröder K, Brandes RP, Siemen D, Ježek P (2020) Glucose-stimulated insulin secretion fundamentally requires H2O2 signaling by NADPH oxidase 4 . Diabetes 69:1341-54. |
Jezek 2018 Antioxid Redox Signal | 2018 | Jezek P, Holendova B, Garlid KD, Jaburek M (2018) Mitochondrial uncoupling proteins: subtle regulators of cellular redox signaling. Antioxid Redox Signal 29:667-714. |
Jezek 2015 Antioxid Redox Signal | 2015 | Ježek J, Dlasková A, Zelenka J, Jabůrek M, Ježek P (2015) H2O2-activated mitochondrial phospholipase iPLA2γ prevents lipotoxic oxidative stress in synergy with UCP2, amplifies signaling via G-protein-coupled receptor GPR40, and regulates insulin secretion in pancreatic β-cells. Antioxid Redox Signal 23:958-72. |
Jaburek 2013 Int J Biochem Cell Biol | 2013 | Jabůrek M, Ježek J, Zelenka J, Ježek P (2013) Antioxidant activity by a synergy of redox-sensitive mitochondrial phospholipase A2 and uncoupling protein-2 in lung and spleen. Int J Biochem Cell Biol 45:816-25. |
Jezek 2010 Physiol Res | 2010 | Ježek J, Jabůrek M, Zelenka J, Ježek P (2010) Mitochondrial phospholipase A2 activated by reactive oxygen species in heart mitochondria induces mild uncoupling. Physiol Res 59:737-47. |
Abstracts
Published | Reference | |
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Jaburek 2014 Abstract MiP2014 | 2014 | The role of phospholipase A2γ in the regulation of mitochondrial uncoupling protein 2–dependent antioxidant function. |
Jezek 2013 Abstract MiP2013 | 2013 | Ježek P, Ježek J, Zelenka J, Dlasková A, Jabůrek M (2013) Antioxidant synergy of mitochondrial uncoupling protein UCP2 and phospholipase iPLA2γ. Mitochondr Physiol Network 18.08. |
Gabrielova 2011 Abstract IOC61 | 2011 | Gabrielova E, Jaburek M, Gazak R, Vostalova J, Jezek J, Kren V, Modriansky M (2011) Evaluation of an oxygen consumption in rat heart mitochondria treatment by polyphenol compounds. MiPNet16.01. |